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Erratum: Ochnaflavone inhibits TNF‐α‐induced human VSMC proliferation via regulation of cell cycle, ERK1/2, and MMP‐9. S.‐J. Suh, U.‐H. Jin, S.‐H. Kim, H.‐W. Chang, J.‐K. Son, S.H. Lee, K.‐H. Son, C.‐H. Kim
Publication year - 2009
Publication title -
journal of cellular biochemistry
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.028
H-Index - 165
eISSN - 1097-4644
pISSN - 0730-2312
DOI - 10.1002/jcb.22257
Subject(s) - oligonucleotide , chemistry , microbiology and biotechnology , matrix metalloproteinase , electrophoretic mobility shift assay , tumor necrosis factor alpha , dna , stereochemistry , biochemistry , transcription factor , biology , immunology , gene
During the corresponding author's transfer from Dongguk University to Sungkyunkwan University in March 2006, data from the previous University was transferred to the corresponding author's new computer. During this data transfer there was a mixing of EMSA data from experiments involving Quercetin (QC), Ochnaflavone (OC), Tanshinone (TS), Crytotanshinone (CT), BMK, and natural extracts. The mixed EMSA data was inadvertently incorporated in more than one publication. Figure 6 has been corrected to new data with re‐confirmation. J. Cell. Biochem. 108: 337, 2009. © 2009 Wiley‐Liss, Inc.6. Effect of OC on the TNF‐α‐induced DNA binding activities of MMP‐9, NF‐κB, and AP‐1 motif in HASMC. Cells were pretreated with indicated OC for 40 min in serum‐free medium, were incubated with TNF‐α (100 ng/ml) for 24 h. Nuclear extracts were analyzed by EMSA for the activated NF‐κB (A) and AP‐1 (B) using radiolabeled oligonucleotide probes, respectively. Indicated values are means of three triplicate experiments.