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A fixed partial epithelial‐mesenchymal transition (EMT) triggers carcinogenesis, whereas asymmetrical division of hybrid EMT cells drives cancer progression
Author(s) -
Denisov Evgeny V.,
Perelmuter Vladimir M.
Publication year - 2018
Publication title -
hepatology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 5.488
H-Index - 361
eISSN - 1527-3350
pISSN - 0270-9139
DOI - 10.1002/hep.29784
Subject(s) - carcinogenesis , biology , epithelial–mesenchymal transition , cancer research , mesenchymal stem cell , epigenetics , cancer , cancer cell , tumor progression , cancer stem cell , neoplastic cell , clone (java method) , phenotype , molecular oncology , metastasis , microbiology and biotechnology , genetics , cell , gene