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The effects of puberty on associations between mood/personality factors and disordered eating symptoms in girls
Author(s) -
Vo Phuong T.,
Fowler Natasha,
Rolan Emily P.,
Culbert Kristen M.,
Racine Sarah E.,
Burt S. Alexandra,
Klump Kelly L.
Publication year - 2021
Publication title -
international journal of eating disorders
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.785
H-Index - 138
eISSN - 1098-108X
pISSN - 0276-3478
DOI - 10.1002/eat.23572
Subject(s) - psychology , mood , personality , anxiety , binge eating , clinical psychology , moderation , disordered eating , eating disorders , psychiatry , social psychology
Objective Negative and positive urgency, anxiety, and depressive symptoms are significant factors of disordered eating (DE) symptoms in early adolescence through young adulthood. However, it is unclear how puberty—a critical developmental milestone that is associated with increased risk for DE symptoms—affects the relationship between these factors and DE symptoms, given that the role of pubertal status has rarely been considered in relation to these associations. Thus, the present study examined whether puberty moderates associations between mood/personality factors and DE in pre‐adolescent and adolescent girls. Method Participants included 981 girls (aged 8–16 years) from the Michigan State University Twin Registry. Mood/personality factors, pubertal status, and DE were assessed with self‐report questionnaires. Results Puberty significantly moderated associations between several factors (negative urgency, positive urgency, trait anxiety, depressive symptoms) and the cognitive symptoms of DE (e.g., shape/weight concerns, body dissatisfaction). Associations between mood/personality factors and cognitive DE were stronger in girls with more advanced pubertal status. By contrast, no significant moderation effects were detected for mood/personality‐dysregulated eating (e.g., binge eating, emotional eating) associations. Discussion Findings identify pubertal development as an important moderator of mood/personality‐DE symptom associations, especially for cognitive DE symptoms that are known to predict the later onset of clinical pathology.