Open AccessUse of cardiac troponin t rapid assay in the diagnosis of a myocardial injury secondary to electrical cardioversion
Author(s) -
Garré Luis,
Álvarez Ángel,
Rubio Mariá,
Pellegrini Alejandro,
Caridi Mariá,
Berardi Alfredo,
Lázzaro Carmen,
Blanco Patricia,
Menehem Claudia,
Díaz MóNica
Publication year - 1997
Publication title -
clinical cardiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.263
H-Index - 72
eISSN - 1932-8737
pISSN - 0160-9289
DOI - 10.1002/clc.4960200707
Subject(s) - medicine , cardioversion , cardiology , electrical cardioversion , troponin i , troponin , myocardial infarction , atrial fibrillation
Background and hypothesis : This study was carried out to determine whether cardiac troponin T test in rapid assay gives positive results in patients previously submitted to cardioversion or electrical defibrillation. Methods : Forty patients with supraventricular tachyarrhythmias lasting no more than 2 days were treated with electrical cardioversion. The total creatine phosphokinase (CPK)‐MB isoenzyme and troponin T in rapid assay were measured at baseline and at 6, 12, and 24 h thereafter. Results : Total CPK baseline levels were normal in all cases; within 4 h, the serum CPK levels increased by 98%, at 6 h by 111.5%, at 12h by 168%, and at 24 h by 225% (p<0.01). The CPK‐MB isoenzyme showed no percentage increase of total CPK higher than 5%, measured at 6, 12, and 24h after the shock, independent of the number of attempts of cardioversion. The troponin T test was also negative in all cases at baseline and at 6, 12, and 24 h after cardioversion. Conclusion : We conclude that the absence of elevations in CPK‐MB levels and cardiac troponin T levels matched clinical and electrocardiographic results showing absence of myocardial damage after electrical cardioversion.