Open AccessNonatherosclerotic causes of coronary artery narrowing—Part III
Author(s) -
Waller Bruce F.,
Fry Edward T. A.,
Hermiller James B.,
Peters Thomas,
Slack John D.
Publication year - 1996
Publication title -
clinical cardiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.263
H-Index - 72
eISSN - 1932-8737
pISSN - 0160-9289
DOI - 10.1002/clc.4960190814
Subject(s) - medicine , cardiology , thrombus , myocardial infarction , kawasaki disease , vasculitis , coronary thrombosis , artery , thrombosis , coronary artery aneurysm , coronary arteries , calcification , coronary artery disease , aneurysm , radiology , disease
Approximately 5% of patients with acute myocardial infarction do not have atherosclerotic coronary artery disease but have other causes for their luminal narrowing. The third part of this three‐part review of nonatherosclerotic causes of coronary narrowing focuses on coronary vasculitis, infectious diseases, Kawasaki's disease, metabolic disorders, metastatic disease, and substance abuse (cocaine). coronary arteritis: (1) focal artery necrosis with or without calcification, (2) acute coronary artery thrombosis or recanal‐ized thrombus unassociated with underlying atherosclerotic plaque, (3) rupture of the vessel wall unassociated with trauma or an interventional procedure, (4) coronary artery wall thickening with secondary luminal narrowing, and (5) wall thinning with aneurysm formation. 3 Specific coronary lesions also may be seen in specific systemic diseases (e.g., pol‐yarteritis nodosa).