Open AccessVariant angina due to deficiency of intracellular magnesium
Author(s) -
Tanabe K.,
Noda K.,
Kamegai M.,
Miyake F.,
Mikawa T.,
Murayama M.,
Sugai J.
Publication year - 1990
Publication title -
clinical cardiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.263
H-Index - 72
eISSN - 1932-8737
pISSN - 0160-9289
DOI - 10.1002/clc.4960130914
Subject(s) - medicine , magnesium , hyperventilation , magnesium deficiency (plants) , ergonovine , angina , cardiology , calcium , st elevation , anesthesia , electrocardiography , myocardial infarction , materials science , metallurgy
A 51‐year‐old man was diagnosed as having variant angina by documentation of typical ST elevation during anginal attack and also by showing coronary arterial spasm (#2 and #12) during hyperventilation on coronary arteriography. Large quantities of calcium blocking agents and nitrates could not improve his symptoms. Lack of intracellular magnesium was suspected from a daily excretion of urine magnesium (5.3 mEq) and magnesium tolerance test (56.7%). After hourly infusion of magnesium sulfate (80 mEq), coronary spasm could not be induced by ergonovine.