Open AccessSerial M‐mode echocardiographic mapping in myocardial infarction: A quantitative evaluation of left ventricular wall motion abnormalities
Author(s) -
Lindvall K.,
Erhardt L.,
Sjögren A.
Publication year - 1983
Publication title -
clinical cardiology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.263
H-Index - 72
eISSN - 1932-8737
pISSN - 0160-9289
DOI - 10.1002/clc.4960060507
Subject(s) - medicine , cardiology , myocardial infarction , anterior wall , posterior wall , infarction
M‐mode echocardiographic (echo) mapping was performed in 44 consecutive patients with a first acute transmural myocardial infarction (AMI). Regional left ventricular wall motion, as reflected by mean systolic wall velocity (V̄), was analyzed in 16 segments on day 1,2, 10, of hospitalization and after one year. Mean systolic wall velocity was significantly reduced (p < 0.05–0.01) in all segments indicated to be infarcted according to ECG. Anteroseptal and anterolateral AMIs showed similar V̄ impairment in anterior segments. In one latero‐apical segment in anterolateral AMI V̄ was significantly lower (p < 0.01), while anteroseptal AMI also showed lower values (p < 0.05) for the midsegment of the middle third of septum. Inferior and inferolateral AMI had similar reductions in V̄ in the inferior segments. In addition depressed wall motion (p < 0.05) was found in one posterior segment in inferior AMI and in two posteroseptal segments in inferolateral AMI. Following the AMI V̄ successively decreased in the 4 central segments in patients with anterior wall infarction during the first 3 days, followed by a significant (p < 0.001) improvement on day 10. No further significant changes were seen after one year. Centrally infarcted segments in inferior AMI showed a minor (p < 0.05) decrease in V̄ from day 1 to day 2, with similar V̄ on day 10. A minor improvement (p < 0.05) was found one year later. Segments adjacent to centrally infarcted segments showed V̄ values similar to infarcted segments during the first 24 hours, followed by rapid improvement in inferior AMI from day 2, with normalization by day 10. Border segments in anterior AMI showed depressed V̄ values during the first 3 days, followed by a normalization by day 10. Noninfarcted segments in patients with inferior AMI showed hyperkinetic wall motion during the first 10 days, with normalization at the one year follow‐up. The changes were moderate though peak V̄ values on days 2 or 3 were significantly higher (p < 0.05) than values one year later. Similarly, wall motion in noninfarcted segments in patients with anterior AMI reached hyperkinetic levels days 3 and 10, which was significantly higher (p < 0.01) than values on days 1, 2, or after one year.