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Middle cerebral artery strokes causing homonymous hemianopia: Positron emission tomography
Author(s) -
Kiyosawa Motohiro,
Bosley Thomas M.,
Kushner Michael,
Jamieson Dara,
Alavi Abass,
Reivich Martin
Publication year - 1990
Publication title -
annals of neurology
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 4.764
H-Index - 296
eISSN - 1531-8249
pISSN - 0364-5134
DOI - 10.1002/ana.410280212
Subject(s) - positron emission tomography , medicine , middle cerebral artery , stroke (engine) , neuroimaging , cortex (anatomy) , cerebral cortex , cerebral hemisphere , fluorodeoxyglucose , lateralization of brain function , nuclear medicine , cardiology , radiology , neuroscience , psychology , ischemia , audiology , psychiatry , mechanical engineering , engineering
Eight patients were evaluated with 18 F‐fluorodeoxyglucose positron emission tomography between 3 and 30 days after isolated stroke involving the middle cerebral artery territory that caused homonymous hemianopia. Diffuse hypometabolism was present throughout the damaged cerebral hemisphere, even in cortical areas not obviously ischemic by clinical examination or neuroimaging. Glucose metabolism in primary and association visual cortex of the damaged hemisphere was decreased by more than 47% ( p < 0.01). Metabolism in the undamaged hemisphere was less profoundly affected, but significant decrements were found in calcarine (40%; p < 0.01) and lateral occipital cortex (35%; p < 0.05).