Hyperprolinemia is not associated with childhood onset schizophrenia | Zendy

Jacquet Hélène | Zendy; Rapoport Judith L. | Zendy; Hecketsweiler Bernadette | Zendy; Bobb Aaron | Zendy; Thibaut Florence | Zendy; Frébourg Thierry | Zendy; Campion Dominique | Zendy

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Hyperprolinemia is not associated with childhood onset schizophrenia

Author(s) -

Jacquet Hélène,

Rapoport Judith L.,

Hecketsweiler Bernadette,

Bobb Aaron,

Thibaut Florence,

Frébourg Thierry,

Campion Dominique

Publication year - 2006

Publication title -

american journal of medical genetics part b: neuropsychiatric genetics

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.393

H-Index - 126

eISSN - 1552-485X

pISSN - 1552-4841

DOI - 10.1002/ajmg.b.30263

Subject(s) - missense mutation , schizoaffective disorder , schizophrenia (object oriented programming) , bipolar disorder , genetics , psychosis , psychology , gene , medicine , mutation , biology , psychiatry , cognition

In a previous report [Jacquet et al., 2005] we have shown that mild to moderate hyperprolinemia resulting from several alterations (either a complete deletion or missense mutations) of the proline dehydrogenase ( PRODH ) gene located on chromosome 22q11 is a risk factor for schizoaffective disorder but not for DSM3 R schizophrenia or bipolar disorder. We now report that hyperprolinemia is not associated with childhood onset schizophrenia (COS). © 2006 Wiley‐Liss, Inc.

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