Premium
Subacute sensory neuropathy associated with Epstein–Barr virus
Author(s) -
Rubin Devon I.,
Daube Jasper R.
Publication year - 1999
Publication title -
muscle and nerve
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.025
H-Index - 145
eISSN - 1097-4598
pISSN - 0148-639X
DOI - 10.1002/(sici)1097-4598(199911)22:11<1607::aid-mus21>3.0.co;2-j
Subject(s) - sensory system , medicine , guillain barre syndrome , polyneuropathy , peripheral neuropathy , spinal cord , sensory loss , sensory nerve , virus , etiology , peripheral nervous system , magnetic resonance imaging , pathology , neuroscience , central nervous system , immunology , surgery , biology , endocrinology , radiology , psychiatry , diabetes mellitus
A 35‐year‐old man experienced severe sensory loss, pseudoathetosis, and areflexia during recovery from a severe viral illness. Sensory nerve action potentials were absent, motor conduction velocities were mildly slowed, and blink reflexes were normal. Magnetic resonance imaging (MRI) revealed abnormal signal within the central and dorsal aspects of the thoracic cord. Acute and convalescent Epstein–Barr virus (EBV) titers suggested EBV as the etiology. Subacute sensory neuropathy, with peripheral and central nervous system involvement, is a rare complication of EBV infection. © 1999 John Wiley & Sons, Inc. Muscle Nerve 22: 1607–1610, 1999