Smad4 Expression in Hepatocellular Carcinoma Differs by Hepatitis Status | Zendy

Lei Yao | Zendy; Fujun Li | Zendy; Zhiqiang Tang | Zendy; Shuang Gao | Zendy; Qe-Quan Wu | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

Smad4 Expression in Hepatocellular Carcinoma Differs by Hepatitis Status

Author(s) -

Lei Yao,

Fujun Li,

Zhiqiang Tang,

Shuang Gao,

Qe-Quan Wu

Publication year - 2012

Publication title -

asian pacific journal of cancer prevention

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.512

H-Index - 75

eISSN - 2476-762X

pISSN - 1513-7368

DOI - 10.7314/apjcp.2012.13.4.1297

Subject(s) - hepatocellular carcinoma , immunohistochemistry , carcinogenesis , biology , viral hepatitis , malignancy , hepatitis c virus , messenger rna , hepatitis b virus , blot , virus , cancer research , pathology , virology , medicine , cancer , gene , immunology , biochemistry , genetics

Primary hepatocellular carcinoma (HCC) is a common malignancy often related to hepatitis viral infection. Smad4 is known to mediate the TGF-β pathway to suppress tumorigenesis. However, the function of Smad4 in HCC is still controversial. In this study we compared levels of Smad4 in HCC tissues with or without hepatitis virus infection and adjacent normal-appearing liver.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research