Does the relief of glucose toxicity act as a mediator in proliferative actions of vanadium on pancreatic islet beta cells in streptozocin diabetic rats? | Zendy

Leila Pirmoradi | Zendy; Mohammad Taghi Mohammadi | Zendy; Akbar Safaei | Zendy; Fakhroddin Mesbah | Zendy; Gholam Abbas Dehghani | Zendy

Open Access

Does the relief of glucose toxicity act as a mediator in proliferative actions of vanadium on pancreatic islet beta cells in streptozocin diabetic rats?

Author(s) -

Leila Pirmoradi,

Mohammad Taghi Mohammadi,

Akbar Safaei,

Fakhroddin Mesbah,

Gholam Abbas Dehghani

Publication year - 2014

Publication title -

pubmed

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.519

H-Index - 31

eISSN - 2008-823X

pISSN - 1028-852X

DOI - 10.6091/ibj.1329.2014

Subject(s) - medicine , endocrinology , diabetes mellitus , islet , insulin , streptozocin , pancreatic islets , toxicity , vanadium , chemistry , streptozotocin , inorganic chemistry

Data shows vanadium protects pancreatic beta cells (BC) from diabetic animals. Whether this effect is direct or through the relief of glucose toxicity is not clear. This study evaluated the potential effect of oral vanadyl sulfate (vanadium) on glycemic status and pancreatic BC of normal and diabetic rats.

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