Open AccessTumor Suppressor DLEC1 can Stimulate the Proliferation of Cancer Cells When AP-2ɑ2 is Down-Regulated in HCT116
Author(s) -
GuoHua Qiu,
Xiaojin Xie,
Linhong Deng,
Shing Chuan Hooi
Publication year - 2015
Publication title -
hepatitis monthly
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.264
H-Index - 37
eISSN - 1735-3408
pISSN - 1735-143X
DOI - 10.5812/hepatmon.29829
Subject(s) - cell cycle , cell growth , gene knockdown , cell cycle checkpoint , transfection , cancer research , small interfering rna , tumor suppressor gene , microbiology and biotechnology , cancer , biology , apoptosis , gene , carcinogenesis , genetics
DLEC1 suppresses tumor cell growth the presence of AP-2α2 and stimulates cell proliferation in the down-regulation of AP-2α2 in DLEC1 over-expression stable clones of HTC116.
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