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Atherosclerosis occurs as a result of low-density lipoprotein (LDL) deposits oxidation. Endothelial dysfunction is an early process of atherosclerosis. Restoring endothelial lining back to normal by endothelial progenitor cells (EPCs) is critical for slowing or reversing vascular disease progression. Oxidative stress from hydrogen peroxide (H 2 O 2 ) is increased in dyslipidemia so that antioxidant agent is required to prevent destruction of blood vessels.

The Inhibitory Effects of Polysaccharide Peptides (PsP) ofGanoderma lucidumagainst Atherosclerosis in Rats with Dyslipidemia