The Contribution of Chemoreceptor-Network Injury to the Development of Respiratory Arrest Following Subarachnoid Hemorrhage | Zendy

Mehmet Dumlu Aydın | Zendy; Atilla Eroğlu | Zendy; Atila Türkyılmaz | Zendy; Alı Fuat Erdem | Zendy; Haci Ahmet Alıcı | Zendy; Nazan Aydın | Zendy; Sare Altaş | Zendy; Bünyami Ünal | Zendy

Open Access

The Contribution of Chemoreceptor-Network Injury to the Development of Respiratory Arrest Following Subarachnoid Hemorrhage

Author(s) -

Mehmet Dumlu Aydın,

Atilla Eroğlu,

Atila Türkyılmaz,

Alı Fuat Erdem,

Haci Ahmet Alıcı,

Nazan Aydın,

Sare Altaş,

Bünyami Ünal

Publication year - 2010

Publication title -

eurasian journal of medicine

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.337

H-Index - 13

eISSN - 1308-8742

pISSN - 1308-8734

DOI - 10.5152/eajm.2010.16

Subject(s) - medicine , carotid body , subarachnoid hemorrhage , anesthesia , respiratory system , brainstem , chemoreceptor , anatomy , surgery , carotid arteries , receptor

Respiratory arrest following brainstem herniation has been attributed to injuries resulting from compression of the respiratory centers. While it is widely perceived that the chemoreceptor network, consisting of the glossopharyngeal nerve and carotid body (GPN-CB), is essential for the modulation of respiration, its contribution to the development of respiratory arrest has not been investigated. Therefore, the aim of this study was to investigate whether injury to the GPN-CB occurs in animals with respiratory arrest caused by experimentally-induced subarachnoid hemorrhage.

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