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Stem Cells-Cancer Research
Author(s) -
Vijaya Krishna Varanasi
Publication year - 2012
Publication title -
journal of carcinogenesis and mutagenesis
Language(s) - English
Resource type - Journals
ISSN - 2157-2518
DOI - 10.4172/2157-2518.s1-e001
Subject(s) - cancer , omics , medicine , bioinformatics , computational biology , biology , physiology
Open access refers to unrestricted access via the internet to articles published in scholarly journals such as “Journal of Carcinogenesis & Mutagenesis”. The main benefit of open access journals is that they are integrated with search engines, such as Google Scholar and anyone or anywhere connected to the web will have free access to their topic of interest. Open Access articles are cited much more than the non open access articles [1] and have greater visibility in the scientific community and public. In medical and pharmaceutical sciences it will enable doctors, pharmacists and clinical researchers to increase their medical knowhow and share it with their colleagues for the advancement of their field of study. OMICS Publishing Group strongly supports this open access initiative and all articles published by OMICS Publishing Group are freely accessible to everyone immediately after publication. Some of the special features of OMICS group journals include digital formatting, audio listening, language translation and ability to share views on articles via social networking. According to a recent report 88% of respondents believed that publicly funded research should be made available to be read and used without barriers to access [2]. The report also revealed that about 94% of cancer researchers use internet for their professional activities every day and sharing of information can accelerate progress in the scientific battle against cancer. The “Journal of Carcinogenesis and Mutagenesis” focuses on the recognition of cellular responses to DNA damage, apoptosis (cell death), including the inactivation of tumor suppressor genes, and analysis of carcinogenic process by genetic and epigenetic alterations in genes for the study of cancer initiation and progression.

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