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We read the recent paper by Unnewher and colleagues ([1][1]) with interest. This paper adds to the growing literature on the role of C5a in human critical illness. The finding of reduced neutrophil CD88 expression in human sepsis was first identified by Furebring and colleagues in 2002 ([2][2]).

Comment on “Changes and Regulation of the C5a Receptor on Neutrophils during Septic Shock in Humans”