Comment on “Inflammasome Activation of IL-18 Results in Endothelial Progenitor Cell Dysfunction in Systemic Lupus Erythematosus” | Zendy

Zhenhua Duan | Zendy; Yujing Zhang | Zendy; Zhen Zeng | Zendy; Faming Pan | Zendy

Open Access

Comment on “Inflammasome Activation of IL-18 Results in Endothelial Progenitor Cell Dysfunction in Systemic Lupus Erythematosus”

Author(s) -

Zhenhua Duan,

Yujing Zhang,

Zhen Zeng,

Faming Pan

Publication year - 2012

Publication title -

the journal of immunology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.737

H-Index - 372

eISSN - 1550-6606

pISSN - 0022-1767

DOI - 10.4049/jimmunol.1290031

Subject(s) - inflammasome , endothelial progenitor cell , progenitor cell , immunology , endothelial dysfunction , medicine , progenitor , endothelial stem cell , biology , microbiology and biotechnology , stem cell , inflammation , genetics , in vitro

Recently, Kahlenberg et al. ([1][1]) found that inhibition of caspase-l disrupted IFN-α–mediated endothelial progenitor cell (EPC)/circulating angiogenic cell (CAC) dysfunction in murine and human systems. It lends more information on the role of IFN-α, which might promote atherosclerosis in

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