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By passive transfer of anti–transglutaminase-3 (TG3) Abs to SCID mice engrafted with normal human skin, Zone et al. ([1][1]) reproduced the immunopathological pattern of dermatitis herpetiformis (DH), confirming the answer to a long-standing question regarding the source of IgA in DH skin.

the journal of immunology/the journal of immunology

Comment on “Dermatitis Herpetiformis Sera or Goat Anti–Transglutaminase-3 Transferred to Human Skin-Grafted Mice Mimics Dermatitis Herpetiformis Immunopathology”