CXCL14 deficiency does not impact the outcome of influenza or Escherichia coli infections in mice | Zendy

Abubaker M. E. Sidahmed | Zendy; Alberto J. León | Zendy; David W. Banner | Zendy; Alyson A. Kelvin | Zendy; Thomas Rowe | Zendy; Ivo Boudakov | Zendy; Norbert Degousse | Zendy; Barry B. Rubin | Zendy; David J. Kelvin | Zendy

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CXCL14 deficiency does not impact the outcome of influenza or Escherichia coli infections in mice

Author(s) -

Abubaker M. E. Sidahmed,

Alberto J. León,

David W. Banner,

Alyson A. Kelvin,

Thomas Rowe,

Ivo Boudakov,

Norbert Degousse,

Barry B. Rubin,

David J. Kelvin

Publication year - 2014

Publication title -

the journal of infection in developing countries

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.322

H-Index - 49

eISSN - 2036-6590

pISSN - 1972-2680

DOI - 10.3855/jidc.3890

Subject(s) - cxcl14 , chemokine , biology , immunology , inflammation , microbiology and biotechnology , phenotype , influenza a virus , cxcl9 , virus , virology , chemokine receptor , gene , genetics

Chemokines are small proteins that regulate different cellular functions, such as leukocyte activation, chemoattraction and inflammation. The chemokine CXCL14 (BRAK) is a highly conserved gene among species and through evolution. It has been shown that CXCL14 is locally upregulated during viral infections, also, it has been found that this chemokine possesses direct antibacterial activities. Nonetheless, the exact role that CXCL14 plays during infection remains elusive.

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