Hemorheological and metabolic consequences of renal ischemia-reperfusion and their modulation by N,N-dimethyl-tryptamine on a rat model | Zendy

Katalin Pető | Zendy; Norbert Németh | Zendy; Anita Mester | Zendy; Zsuzsanna Magyar | Zendy; Souleiman Ghanem | Zendy; Viktória Somogyi | Zendy; Bence Tánczos | Zendy; Ádám Deák | Zendy; László Bidiga | Zendy; Ede Frecska | Zendy; Balázs Nemes | Zendy

Open Access

Hemorheological and metabolic consequences of renal ischemia-reperfusion and their modulation by N,N-dimethyl-tryptamine on a rat model

Author(s) -

Katalin Pető,

Norbert Németh,

Anita Mester,

Zsuzsanna Magyar,

Souleiman Ghanem,

Viktória Somogyi,

Bence Tánczos,

Ádám Deák,

László Bidiga,

Ede Frecska,

Balázs Nemes

Publication year - 2018

Publication title -

clinical hemorheology and microcirculation

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 0.546

H-Index - 54

eISSN - 1875-8622

pISSN - 1386-0291

DOI - 10.3233/ch-170361

Subject(s) - ischemia , erythrocyte deformability , renal ischemia , medicine , kidney , red blood cell , agonist , erythrocyte aggregation , endocrinology , renal circulation , chemistry , reperfusion injury , pharmacology , receptor , renal blood flow , hematocrit

Micro-rheological relations of renal ischemia-reperfusion (I/R) have not been completely elucidated yet. Concerning anti-inflammatory agents, it is supposed that sigma-1 receptor agonist N,N-dimethyl-tryptamin (DMT) can be useful to reduce I/R injury.

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