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In a recent Diabetes article, Biscetti et al. (1) report that high-mobility group box-1 (HMGB1) protein levels are reduced in the skeletal muscle of diabetic mice and that exogenous HMGB1 administration, following the induction of hindlimb ischemia, promotes angiogenesis through a VEGF-dependent mechanism.With regard to the angiogenic effect of HMGB1, we have previously demonstrated that both HMGB1 administration and …

diabetes

Comment on: Biscetti et al. (2010) High-Mobility Group Box-1 Protein Promotes Angiogenesis After Peripheral Ischemia in Diabetic Mice Through a VEGF-Dependent Mechanism. Diabetes;59:1496–1505