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Spontaneous bacterial peritonitis (SBP) is a major complication of liver   cirrhosis, which is associated with increased mortality. While recent studies   have demonstrated the involvement of reactive oxygen species in the   pathogenesis of liver cirrhosis, the role of oxidative stress in the   development of SBP has not yet been completely established. The present study   aims to evaluate the role of oxidative stress in the pathogenesis of this   complication and also the relevance of the specific treatment on these   aspects. We present here some of our preliminary results regarding the   specific activity of glutathione peroxidase (GPX), a very important   antioxidant enzyme, from both serum and ascitic fluid of patients with   decompensated cirrhosis and SBP, patients diagnosed with decompensated liver   cirrhosis with ascites and patients with compensated liver cirrhosis. Our   results demonstrate the presence of an increased oxidative stress in patients   with decompensated cirrhosis and SBP compared with those without SBS and   those with compensated liver cirrhosis, as demonstrated through the   significant decrease of the specific activity of GPX. The measurement of   these oxidative stress parameters may have an important role in the diagnosis   and follow-up of this important liver pathology and the auxiliary treatment

A study of the levels of glutathione peroxidase in patients with spontaneous bacterial peritonitis