Urinary Hepcidin-25 and Risk of Acute Kidney Injury Following Cardiopulmonary Bypass | Zendy

Julie Ho | Zendy; Martina Reslerova | Zendy; Brent Gali | Zendy; Ang Gao | Zendy; Jennifer Bestland | Zendy; David N. Rush | Zendy; Peter Nickerson | Zendy; Claudio Rigatto | Zendy

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Urinary Hepcidin-25 and Risk of Acute Kidney Injury Following Cardiopulmonary Bypass

Author(s) -

Julie Ho,

Martina Reslerova,

Brent Gali,

Ang Gao,

Jennifer Bestland,

David N. Rush,

Peter Nickerson,

Claudio Rigatto

Publication year - 2011

Publication title -

clinical journal of the american society of nephrology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.755

H-Index - 151

eISSN - 1555-905X

pISSN - 1555-9041

DOI - 10.2215/cjn.01000211

Subject(s) - hepcidin , medicine , acute kidney injury , cardiopulmonary bypass , urinary system , urology , intensive care unit , univariate analysis , prospective cohort study , gastroenterology , cardiology , multivariate analysis , inflammation

Acute kidney injury (AKI) complicating cardiopulmonary bypass (CPB) results in increased morbidity and mortality. Urinary hepcidin-25 has been shown to be elevated in patients who do not develop AKI after CPB using semiquantitative mass spectrometry (SELDI TOF-MS). The goals of this study were to quantitatively validate these findings with ELISA and evaluate the diagnostic performance of hepcidin-25 for AKI.

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