Arsenic trioxide-mediated oxidative stress and genotoxicity in human hepatocellular carcinoma cells | Zendy

Daoud Ali | Zendy; Saad Alkahtani | Zendy; Maqsood A. Siddiqui | Zendy; Saud Alarifi | Zendy; Bahy A. Ali | Zendy

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Arsenic trioxide-mediated oxidative stress and genotoxicity in human hepatocellular carcinoma cells

Author(s) -

Daoud Ali,

Saad Alkahtani,

Maqsood A. Siddiqui,

Saud Alarifi,

Bahy A. Ali

Publication year - 2013

Publication title -

oncotargets and therapy

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.054

H-Index - 60

ISSN - 1178-6930

DOI - 10.2147/ott.s38227

Subject(s) - arsenic trioxide , oxidative stress , chemistry , genotoxicity , reactive oxygen species , hepatocellular carcinoma , superoxide dismutase , apoptosis , lactate dehydrogenase , arsenic , arsenic toxicity , catalase , pharmacology , microbiology and biotechnology , cancer research , medicine , biochemistry , biology , toxicity , enzyme , organic chemistry

Arsenic is a ubiquitous environmental toxicant, and abnormalities of the skin, lung, kidney, and liver are the most common outcomes of long-term arsenic exposure. This study was designed to investigate the possible mechanisms of genotoxicity induced by arsenic trioxide in human hepatocellular carcinoma cells.

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