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Accumulating evidence suggests that the abnormal expression of the circadian clock gene PER1 is closely related to the development and progression of cancer. However, the exact molecular mechanism by which the abnormal expression of PER1 induces carcinogenesis is unclear. This study was conducted to investigate the alterations in downstream cell cycle genes, cell cycle distribution, cell proliferation, apoptosis, and in vivo tumorigenicity in SCC15 oral squamous cell carcinoma cells after PER1 downregulation.

oncotargets and therapy

The important tumor suppressor role of PER1 in regulating the cyclin&amp;ndash;CDK&amp;ndash;CKI network in SCC15 human oral squamous cell carcinoma cells