Silencing of DHX32 increases the proliferation of liver cancer cells
Author(s) -
Minjing Cai,
Jianhui Zhu,
Jianquan He,
Zhongying Zhang,
Xianming Liang
Publication year - 2020
Publication title -
translational cancer research
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 0.254
H-Index - 30
eISSN - 2219-6803
pISSN - 2218-676X
DOI - 10.21037/tcr.2020.02.35
Subject(s) - liver cancer , cancer , gene knockdown , cancer research , cancer cell , medicine , kinase , pathology , biology , apoptosis , microbiology and biotechnology , biochemistry
Liver cancer ranks fifth in malignancy incidence throughout the world (1) and is the second leading cause of cancerrelated death in China (2). Chronic hepatitis B or C infection and alcohol abuse have been identified to be the major risk factors for liver cancer development (3,4), which is characterized by its high complexity and heterogeneity. RNA helicases, as motor proteins, modulate the Original Article
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