The potential of glioma-associated oncogene homolog 1 (GLI1) as a therapeutic target in endometriosis

Department of Obstetrics and Gynecology, University Hospital and University of Bern, Bern, Switzerland; Department of Obstetrics and Gynecology, School for Oncology and Developmental Biology (GROW), Maastricht University Medical Center, Maastricht, The Netherlands Correspondence to: Konstantinos Nirgianakis, MD. University Hospital Bern, Friedbühlstrasse 19 CH-3010 Bern, Switzerland. Email: konstantinos.nirgianakis@insel.ch. Provenance and Peer Review: This article was commissioned by the Editorial Office, Annals of Translational Medicine. The article did not undergo external peer review. Comment on: Liu H, Zhang W, Wang L, et al. GLI1 is increased in ovarian endometriosis and regulates migration, invasion and proliferation of human endometrial stromal cells in endometriosis. Ann Transl Med 2019;7:663.