Open AccessPPAR-δ is repressed in Huntington's disease, is required for normal neuronal function and can be targeted therapeutically
Author(s) -
Audrey S. Dickey,
Victor V. Pineda,
Taiji Tsunemi,
Patrick P. Liu,
Helen C. Miranda,
Stephen K. Gilmore-Hall,
Nicole Lomas,
Kunal R. Sampat,
Anne Buttgereit,
Mark-Joseph Manalang Torres,
April L. Flores,
Martin Arreola Villanueva,
Nicolas Arbez,
Sergey Akimov,
Terry Gaasterland,
Eduardo R. Lazarowski,
Christopher A. Ross,
G Yeo,
Bryce L. Sopher,
Gavin Magnuson,
Anthony B. Pinkerton,
Eliezer Masliah,
Albert R. La Spada
Publication year - 2016
Publication title -
carolina digital repository (university of north carolina at chapel hill)
Language(s) - English
DOI - 10.17615/w1ba-cf62
Subject(s) - huntington's disease , neuroscience , disease , function (biology) , neurodegeneration , gain of function , medicine , psychology , biology , genetics , mutation , gene
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