Clusters Not Classifications: Making Sense of Complement-Mediated Kidney Injury | Zendy

H. Terence Cook | Zendy; Matthew C. Pickering | Zendy

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Clusters Not Classifications: Making Sense of Complement-Mediated Kidney Injury

Author(s) -

H. Terence Cook,

Matthew C. Pickering

Publication year - 2017

Publication title -

journal of the american society of nephrology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 4.451

H-Index - 279

eISSN - 1533-3450

pISSN - 1046-6673

DOI - 10.1681/asn.2017111183

Subject(s) - alternative complement pathway , glomerulopathy , complement system , regulator , atypical hemolytic uremic syndrome , membranoproliferative glomerulonephritis , disease , complement factor b , complement (music) , factor h , pathology , biology , immunology , medicine , kidney , microbiology and biotechnology , glomerulonephritis , genetics , immune system , phenotype , gene , complementation

In 2010, we suggested the name C3 glomerulopathy to encompass a group of glomerular diseases characterized by the presence of glomerular C3 in the absence of substantial Ig and without deposition of the early components of the classic or lectin pathways of complement activation.[1][1] Within this

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