Nitric oxide mediates abnormal responsiveness of thyroid arteries in methimazole-treated patients | Zendy

Joaquín Ortega | Zendy; José M. Vila | Zendy; María Dolores Mauricio | Zendy; Gloria Segarra | Zendy; Pascual Medina | Zendy; Juan Martínez-León | Zendy; Salvador Lluch | Zendy

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Nitric oxide mediates abnormal responsiveness of thyroid arteries in methimazole-treated patients

Author(s) -

Joaquín Ortega,

José M. Vila,

María Dolores Mauricio,

Gloria Segarra,

Pascual Medina,

Juan Martínez-León,

Salvador Lluch

Publication year - 2005

Publication title -

european journal of endocrinology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.897

H-Index - 148

eISSN - 1479-683X

pISSN - 0804-4643

DOI - 10.1530/eje.1.01889

Subject(s) - euthyroid , endocrinology , medicine , acetylcholine , charybdotoxin , apamin , contraction (grammar) , vasodilation , nitric oxide , inferior thyroid artery , propylthiouracil , chemistry , thyroid , potassium channel , recurrent laryngeal nerve

We studied the intervention of nitric oxide (NO), prostacyclin and endothelium-derived hyperpolarizing factor (EDHF) in mediating responses to acetylcholine in thyroid arteries from euthyroid and methimazole-treated (MT) patients.

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