Differential effects of all-trans-retinoic acid (RA) on Erk1/2 phosphorylation and cAMP accumulation in normal and malignant human prostate epithelial cells: Erk1/2 inhibition restores RA-induced decrease of cell growth in malignant prostate cells | Zendy

Daniela Pasquali | Zendy; Paolo Chieffi | Zendy; William J. Deery | Zendy; Gianfranco Nicoletti | Zendy; Antonio Bellastella | Zendy; Antonio Agostino Sinisi | Zendy

Open Access

Differential effects of all-trans-retinoic acid (RA) on Erk1/2 phosphorylation and cAMP accumulation in normal and malignant human prostate epithelial cells: Erk1/2 inhibition restores RA-induced decrease of cell growth in malignant prostate cells

Author(s) -

Daniela Pasquali,

Paolo Chieffi,

William J. Deery,

Gianfranco Nicoletti,

Antonio Bellastella,

Antonio Agostino Sinisi

Publication year - 2005

Publication title -

european journal of endocrinology

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.897

H-Index - 148

eISSN - 1479-683X

pISSN - 0804-4643

DOI - 10.1530/eje.1.01875

Subject(s) - retinoic acid , endocrinology , medicine , phosphorylation , prostate , cell growth , differential effects , cell , cancer research , chemistry , biology , microbiology and biotechnology , biochemistry , gene , cancer

All-trans-retinoic acid (RA) regulates cellular growth, differentiation and apoptosis in human prostate by binding to RA receptors. Non-genomic retinoid effects on signal transduction kinases in the cytoplasm are also described in several cells but they are still unknown in prostate cells.

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