Prenatal exposure to cigarette smoke interacts with OPRM1 to modulate dietary preference for fat | Zendy

Ken W. K. Lee | Zendy; Michał Abrahamowicz | Zendy; Gabriel Leonard | Zendy; Louis Richer | Zendy; Michel Perron | Zendy; Suzanne Veillette | Zendy; Eva Reischl | Zendy; Luigi Bouchard | Zendy; Daniel Gaudet | Zendy; Tomáš Paus | Zendy; Zdenka Pausová | Zendy

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Prenatal exposure to cigarette smoke interacts with OPRM1 to modulate dietary preference for fat

Author(s) -

Ken W. K. Lee,

Michał Abrahamowicz,

Gabriel Leonard,

Louis Richer,

Michel Perron,

Suzanne Veillette,

Eva Reischl,

Luigi Bouchard,

Daniel Gaudet,

Tomáš Paus,

Zdenka Pausová

Publication year - 2015

Publication title -

journal of psychiatry and neuroscience

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.767

H-Index - 99

eISSN - 1488-2434

pISSN - 1180-4882

DOI - 10.1503/jpn.130263

Subject(s) - medicine , epigenetics , dna methylation , population , pregnancy , physiology , endocrinology , bioinformatics , genetics , biology , gene , environmental health , gene expression

Preference for fatty foods is a risk factor for obesity. It is a complex behaviour that involves the brain reward system and is regulated by genetic and environmental factors, such as the opioid receptor mu-1 gene (OPRM1) and prenatal exposure to maternal cigarette smoking (PEMCS). We examined whether OPRM1 and PEMCS interact in influencing fat intake and whether exposure-associated epigenetic modifications of OPRM1 may mediate this gene-environment interaction.

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