Interpreting Cardiac Troponin Results from High-Sensitivity Assays in Chronic Kidney Disease without Acute Coronary Syndrome | Zendy

Christopher R. deFilippi | Zendy; Stephen L. Seliger | Zendy; Walter E. Kelley | Zendy; ShowHong Duh | Zendy; Michael K. Hise | Zendy; Robert H. Christenson | Zendy; Myles Wolf | Zendy; Hanna K. Gaggin | Zendy; James L. Januzzi | Zendy

Open Access

Interpreting Cardiac Troponin Results from High-Sensitivity Assays in Chronic Kidney Disease without Acute Coronary Syndrome

Author(s) -

Christopher R. deFilippi,

Stephen L. Seliger,

Walter E. Kelley,

ShowHong Duh,

Michael K. Hise,

Robert H. Christenson,

Myles Wolf,

Hanna K. Gaggin,

James L. Januzzi

Publication year - 2012

Publication title -

clinical chemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.705

H-Index - 218

eISSN - 1530-8561

pISSN - 0009-9147

DOI - 10.1373/clinchem.2012.185322

Subject(s) - medicine , cardiology , interquartile range , renal function , kidney disease , ejection fraction , myocardial infarction , acute coronary syndrome , coronary artery disease , population , troponin i , creatinine , heart failure , environmental health

Quantification and comparison of high-sensitivity (hs) cardiac troponin I (cTnI) and cTnT concentrations in chronic kidney disease (CKD) have not been reported. We examined the associations between hs cTnI and cTnT, cardiovascular disease, and renal function in outpatients with stable CKD.

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