Increases of Cardiac Troponin in Conditions other than Acute Coronary Syndrome and Heart Failure
Author(s) -
Walter E. Kelley,
James L. Januzzi,
Robert H. Christenson
Publication year - 2009
Publication title -
clinical chemistry
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 1.705
H-Index - 218
eISSN - 1530-8561
pISSN - 0009-9147
DOI - 10.1373/clinchem.2009.130799
Subject(s) - medicine , acute coronary syndrome , heart failure , cardiology , troponin , intensive care unit , coronary artery disease , intensive care medicine , myocardial infarction
Although cardiac troponin (cTn) is a cornerstone marker in the assessment and management of patients with acute coronary syndrome (ACS) and heart failure (HF), cTn is not diagnostically specific for any single myocardial disease process. This narrative review discusses increases in cTn that result from acute and chronic diseases, iatrogenic causes, and myocardial injury other than ACS and HF.
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