Hyperhomocysteinemia, Endothelial Nitric Oxide Synthase Polymorphism, and Risk of Coronary Artery Disease | Zendy

Mohsen Kerkeni | Zendy; Faouzi Addad | Zendy; Maryline Chauffert | Zendy; Anne Myara | Zendy; Mohamed Ben Farhat | Zendy; Abdelhédi Miled | Zendy; Khira Maaroufi | Zendy; F Trivin | Zendy

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Hyperhomocysteinemia, Endothelial Nitric Oxide Synthase Polymorphism, and Risk of Coronary Artery Disease

Author(s) -

Mohsen Kerkeni,

Faouzi Addad,

Maryline Chauffert,

Anne Myara,

Mohamed Ben Farhat,

Abdelhédi Miled,

Khira Maaroufi,

F Trivin

Publication year - 2005

Publication title -

clinical chemistry

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 1.705

H-Index - 218

eISSN - 1530-8561

pISSN - 0009-9147

DOI - 10.1373/clinchem.2005.057950

Subject(s) - hyperhomocysteinemia , enos , coronary artery disease , homocysteine , medicine , genotype , gastroenterology , biology , endocrinology , cardiology , nitric oxide synthase , nitric oxide , genetics , gene

Hyperhomocysteinemia is an independent, graded risk factor for coronary artery disease (CAD). The G894T variant of endothelial nitric oxide synthase (eNOS) was postulated to be associated with hyperhomocysteinemia and could influence individual susceptibility to CAD. The aims of this study were to investigate (a) the relationship of the eNOS G894T polymorphism with the presence and the severity of CAD and (b) the possible relationship between hyperhomocysteinemia and the eNOS G894T variant for the risk of CAD severity in a Tunisian population.

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