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Nitric oxide (NO) plays a relevant role in various events during atherogenesis. In vitro data suggest that NO may modulate homocysteine (Hcy) concentrations. The aim of this study was to investigate the role of endothelial nitric oxide synthase (eNOS) -786T&gt;C, 894G&gt;T, and 4a4b polymorphisms in influencing Hcy concentrations.

clinical chemistry

Endothelial Nitric Oxide Synthase −786T&amp;gt;C, but Not 894G&amp;gt;T and 4a4b, Polymorphism Influences Plasma Homocysteine Concentrations in Persons with Normal Vitamin Status