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We previously reported that mutations occurred in the gene myosin5 were responsible for resistance to the fungicide phenamacril in  Fusarium graminearum . Here, we determined whether there is a functional link between phenamacril resistance and the myosin proteins FaMyo2B and Famyo2 in  Fusarium asiaticum , which is the major causal agent of Fusarium head blight in China. We found that  FaMyo2B  acts jointly with  FaSmy1  to affect resistance to phenamacril in  F .  asiaticum . We also found that  FaMyo2B  disruption mutant and  Famyo2  deletion mutant were defective in hyphal branching, conidiation, and sexual reproduction. Δ Famyo2  also had an enhanced sensitivity to cell wall damaging agents and an abnormal distribution of septa and nuclei. In addition, the  FaMyo2B  and  Famyo2  mutants had reduced pathogenicity on wheat coleoptiles and flowering wheat heads. Taken together, these results reveal that  FaMyo2B  and  Famyo2  are required for several  F .  asiaticum  developmental processes and activities, which help us better understand the resistance mechanism and find the most effective approach to control FHB.

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Myosins FaMyo2B and Famyo2 Affect Asexual and Sexual Development, Reduces Pathogenicity, and FaMyo2B Acts Jointly with the Myosin Passenger Protein FaSmy1 to Affect Resistance to Phenamacril in Fusarium asiaticum