Prenatal Air Pollution Exposures, DNA Methyl Transferase Genotypes, and Associations with Newborn LINE1 and Alu Methylation and Childhood Blood Pressure and Carotid Intima-Media Thickness in the Children’s Health Study | Zendy

Carrie V. Breton | Zendy; Jin Yao | Zendy; Josh Millstein | Zendy; Lu Gao | Zendy; Kimberly D. Siegmund | Zendy; Wendy J. Mack | Zendy; Lora Whitfield-Maxwell | Zendy; Fred Lurmann | Zendy; Howard N. Hodis | Zendy; Ed Avol | Zendy; Frank D. Gilliland | Zendy

Open Access

Prenatal Air Pollution Exposures, DNA Methyl Transferase Genotypes, and Associations with Newborn LINE1 and Alu Methylation and Childhood Blood Pressure and Carotid Intima-Media Thickness in the Children’s Health Study

Author(s) -

Carrie V. Breton,

Jin Yao,

Josh Millstein,

Lu Gao,

Kimberly D. Siegmund,

Wendy J. Mack,

Lora Whitfield-Maxwell,

Fred Lurmann,

Howard N. Hodis,

Ed Avol,

Frank D. Gilliland

Publication year - 2016

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp181

Subject(s) - dna methylation , epigenetics , dnmt3b , methylation , pregnancy , blood pressure , medicine , methyltransferase , genotype , genetics , biology , physiology , gene , gene expression

Although exposure to ambient air pollutants increases cardiovascular disease risk in adults little is known about the effects of prenatal exposure. Genetic variation and epigenetic alterations are two mechanisms that may influence the effects of early-life exposures on cardiovascular phenotypes.

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