Neonatal Exposure to Brominated Flame Retardant BDE-47 Reduces Long-Term Potentiation and Postsynaptic Protein Levels in Mouse Hippocampus | Zendy

Milou M.L. Dingemans | Zendy; Geert M. J. Ramakers | Zendy; Fabrizio Gardoni | Zendy; Regina G.D.M. van Kleef | Zendy; Åke Bergman | Zendy; Mónica Di Luca | Zendy; Martin van den Berg | Zendy; Remco H.S. Westerink | Zendy; Henk P.M. Vijverberg | Zendy

Open Access

Neonatal Exposure to Brominated Flame Retardant BDE-47 Reduces Long-Term Potentiation and Postsynaptic Protein Levels in Mouse Hippocampus

Author(s) -

Milou M.L. Dingemans,

Geert M. J. Ramakers,

Fabrizio Gardoni,

Regina G.D.M. van Kleef,

Åke Bergman,

Mónica Di Luca,

Martin van den Berg,

Remco H.S. Westerink,

Henk P.M. Vijverberg

Publication year - 2007

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.9860

Subject(s) - long term potentiation , postsynaptic potential , synaptic plasticity , excitatory postsynaptic potential , neurotransmission , chemistry , nmda receptor , hippocampal formation , glutamate receptor , endocrinology , medicine , biology , biochemistry , receptor , inhibitory postsynaptic potential

Increasing environmental levels of brominated flame retardants raise concern about possible adverse effects, particularly through early developmental exposure.

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