Elevation of Cellular BPDE Uptake by Human Cells: A Possible Factor Contributing to Co-Carcinogenicity by Arsenite | Zendy

Shengwen Shen | Zendy; Jane Lee | Zendy; Xuejun Sun | Zendy; Hailin Wang | Zendy; Michael Weinfeld | Zendy; X. Chris Le | Zendy

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Elevation of Cellular BPDE Uptake by Human Cells: A Possible Factor Contributing to Co-Carcinogenicity by Arsenite

Author(s) -

Shengwen Shen,

Jane Lee,

Xuejun Sun,

Hailin Wang,

Michael Weinfeld,

X. Chris Le

Publication year - 2006

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.9284

Subject(s) - carcinogen , benzo(a)pyrene , dna damage , chemistry , dna repair , dna , chromatin , arsenite , arsenic , glutathione , nucleotide excision repair , sodium arsenite , biochemistry , enzyme , organic chemistry

Arsenite (iAsIII) can promote mutagenicity and carcinogenicity of other carcinogens. Considerable attention has focused on interference with DNA repair by inorganic arsenic, especially the nucleotide excision repair (NER) pathway, whereas less is known about the effect of arsenic on the induction of DNA damage by other agents.

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