Does aluminum lactate cause spinal cord infarction in rabbits?

as potential toxicants to rabbits (1). As stated by the authors, the role of aluminum in central nervous system diseases (Alzheimer's disease, dialysis dementia) is currently under intensive investigation. Therefore, my attention was drawn to their report of "posterior paraplegia with a wide infarcted area in the spinal cord" in two ofthree rabbits that had been injected intravenously with aluminum lactate in the form ofa suspension ofliposomes. It should be noted that no spinal cord infarct was found in the third rabbit, although it had received a much larger amount of inoculum, or in any ofsix rabbits that had been injected with much larger amounts ofaluminum lactate as an aqueous solution. The authors hypothesize ischemia as an etiologic factor. They might have been thinking ofembolic phenomena related to the injected suspension of lipid material. However, emboli might then have been expected in the brain even more than in the spinal cord, and especially in the third rabbit that received a much larger dose. I would like to offer the alternative suggestion that the spinal cord infarcts may have been caused by accidental traumatic vertebral fracture or dislocation. It is known that sudden kicking or jumping movements by the rabbit's powerful hindlimbs