Placental DNA Methylation Related to Both Infant Toenail Mercury and Adverse Neurobehavioral Outcomes | Zendy

Jennifer Z.J. Maccani | Zendy; Devin C. Koestler | Zendy; Barry M. Lester | Zendy; E. Andrés Houseman | Zendy; David A. Armstrong | Zendy; Karl T. Kelsey | Zendy; Carmen J. Marsit | Zendy

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Placental DNA Methylation Related to Both Infant Toenail Mercury and Adverse Neurobehavioral Outcomes

Author(s) -

Jennifer Z.J. Maccani,

Devin C. Koestler,

Barry M. Lester,

E. Andrés Houseman,

David A. Armstrong,

Karl T. Kelsey,

Carmen J. Marsit

Publication year - 2015

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.1408561

Subject(s) - mercury (programming language) , dna methylation , methylation , mercury exposure , adverse effect , methylmercury , medicine , pregnancy , biology , environmental health , genetics , dna , gene , ecology , biomonitoring , computer science , bioaccumulation , gene expression , programming language

Prenatal mercury (Hg) exposure is associated with adverse child neurobehavioral outcomes. Because Hg can interfere with placental functioning and cross the placenta to target the fetal brain, prenatal Hg exposure can inhibit fetal growth and development directly and indirectly.

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