Chronic Oral Exposure to Bisphenol A Results in a Nonmonotonic Dose Response in Mammary Carcinogenesis and Metastasis in MMTV-erbB2 Mice | Zendy

Sarah Jenkins | Zendy; Jun Wang | Zendy; Isam A. Eltoum | Zendy; Renée A. Desmond | Zendy; Coral A. Lamartiniere | Zendy

Open Access

Chronic Oral Exposure to Bisphenol A Results in a Nonmonotonic Dose Response in Mammary Carcinogenesis and Metastasis in MMTV-erbB2 Mice

Author(s) -

Sarah Jenkins,

Jun Wang,

Isam A. Eltoum,

Renée A. Desmond,

Coral A. Lamartiniere

Publication year - 2011

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.1103850

Subject(s) - carcinogenesis , mammary gland , mammary tumor , carcinogen , endocrinology , medicine , proliferation index , mouse mammary tumor virus , metastasis , cancer research , apoptosis , xenoestrogen , cell growth , biology , immunohistochemistry , cancer , breast cancer , estrogen receptor , immunology , biochemistry , virus

Bisphenol A (BPA) is a synthetic compound used to produce plastics and epoxy resins. BPA can leach from these products in appreciable amounts, resulting in nearly ubiquitous daily exposure to humans. Whether BPA is harmful to humans, especially when administered orally in concentrations relevant to humans, is a topic of debate.

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