Aryl Hydrocarbon Receptor Deficiency Enhances Insulin Sensitivity and Reduces PPAR-α Pathway Activity in Mice | Zendy

Chun Wang | Zendy; Canxin Xu | Zendy; Stacey L. Krager | Zendy; Kathleen M. Bottum | Zendy; DuanFang Liao | Zendy; Shelley A. Tischkau | Zendy

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Aryl Hydrocarbon Receptor Deficiency Enhances Insulin Sensitivity and Reduces PPAR-α Pathway Activity in Mice

Author(s) -

Chun Wang,

Canxin Xu,

Stacey L. Krager,

Kathleen M. Bottum,

DuanFang Liao,

Shelley A. Tischkau

Publication year - 2011

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.1103593

Subject(s) - aryl hydrocarbon receptor , medicine , endocrinology , peroxisome proliferator activated receptor , carbohydrate metabolism , insulin resistance , glucose homeostasis , biology , fatty acid metabolism , signal transduction , chemistry , receptor , metabolism , insulin , biochemistry , transcription factor , gene

Numerous man-made pollutants activate the aryl hydrocarbon receptor (AhR) and are risk factors for type 2 diabetes. AhR signaling also affects molecular clock genes to influence glucose metabolism.

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