PI-3K/Akt Pathway-Dependent Cyclin D1 Expression Is Responsible for Arsenite-Induced Human Keratinocyte Transformation | Zendy

Weiming Ouyang | Zendy; Wenjing Luo | Zendy; Dongyun Zhang | Zendy; Jinlong Jian | Zendy; Qian Ma | Zendy; Jingxia Li | Zendy; Xianglin Shi | Zendy; Jingyuan Chen | Zendy; Jimin Gao | Zendy; Chuanshu Huang | Zendy

AI Assistant Blog Pricing

Home ZAIA Blog

Open Access

PI-3K/Akt Pathway-Dependent Cyclin D1 Expression Is Responsible for Arsenite-Induced Human Keratinocyte Transformation

Author(s) -

Weiming Ouyang,

Wenjing Luo,

Dongyun Zhang,

Jinlong Jian,

Qian Ma,

Jingxia Li,

Xianglin Shi,

Jingyuan Chen,

Jimin Gao,

Chuanshu Huang

Publication year - 2007

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.10403

Subject(s) - hacat , cyclin d1 , arsenite , protein kinase b , cyclin d , carcinogenesis , gene knockdown , cancer research , cell growth , biology , pi3k/akt/mtor pathway , cyclin a , microbiology and biotechnology , chemistry , cell culture , cell , signal transduction , cell cycle , biochemistry , cancer , genetics , arsenic , organic chemistry

Long-term exposure of arsenite leads to human skin cancer. However, the exact mechanisms of arsenite-induced human skin carcinogenesis remain to be defined.

The content you want is available to Zendy users.

Already have an account? Click here to sign in.

Having issues? You can contact us here

Accelerating Research