Perinatal Exposure to Environmentally Relevant Levels of Bisphenol A Decreases Fertility and Fecundity in CD-1 Mice
Author(s) -
Nicolas J. Cabaton,
Perinaaz R. Wadia,
Beverly S. Rubin,
Daniel Zalko,
Cheryl M. Schaeberle,
Michael H. Askenase,
Jennifer L. Gadbois,
Andrew P. Tharp,
Gregory S. Whitt,
Carlos Sonnenschein,
Ana M. Soto
Publication year - 2010
Publication title -
environmental health perspectives
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.257
H-Index - 282
eISSN - 1552-9924
pISSN - 0091-6765
DOI - 10.1289/ehp.1002559
Subject(s) - fecundity , litter , estrous cycle , diethylstilbestrol , biology , lactation , offspring , benzhydryl compounds , endocrinology , fertility , gestation , pregnancy , reproduction , sexual maturity , medicine , bisphenol a , ovary , physiology , estrogen , chemistry , population , ecology , genetics , epoxy , environmental health , organic chemistry , agronomy
Perinatal exposure to low-doses of bisphenol A (BPA) results in alterations in the ovary, uterus, and mammary glands and in a sexually dimorphic region of the brain known to be important for estrous cyclicity.
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