Prenatal and Early, but Not Late, Postnatal Exposure of Mice to Sidestream Tobacco Smoke Increases Airway Hyperresponsiveness Later in Life | Zendy

Zhong-Xin Wu | Zendy; Dawn D. Hunter | Zendy; Vincent Kish | Zendy; Katherine M. Benders | Zendy; Thomas Batchelor | Zendy; Richard D. Dey | Zendy

Open Access

Prenatal and Early, but Not Late, Postnatal Exposure of Mice to Sidestream Tobacco Smoke Increases Airway Hyperresponsiveness Later in Life

Author(s) -

Zhong-Xin Wu,

Dawn D. Hunter,

Vincent Kish,

Katherine M. Benders,

Thomas Batchelor,

Richard D. Dey

Publication year - 2009

Publication title -

environmental health perspectives

Language(s) - English

Resource type - Journals

SCImago Journal Rank - 2.257

H-Index - 282

eISSN - 1552-9924

pISSN - 0091-6765

DOI - 10.1289/ehp.0800511

Subject(s) - methacholine , medicine , gestation , in utero , tobacco smoke , inhalation , inhalation exposure , respiratory system , fetus , physiology , endocrinology , lung , nerve growth factor , pregnancy , respiratory disease , anesthesia , biology , receptor , environmental health , genetics

Cigarette smoke exposure in utero and during early postnatal development increases the incidence of asthma and airway hyperresponsiveness (AHR) later in life, suggesting that a possible critical period of developmental sensitivity exists in the prenatal and early postnatal periods.

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