Open AccessProgression of endometriosis to cancer: too MUCh FoxP3+ regulatory T-cell response?
Author(s) -
G. Aleph Prieto
Publication year - 2011
Publication title -
disease models and mechanisms
Language(s) - English
Resource type - Journals
SCImago Journal Rank - 2.327
H-Index - 83
eISSN - 1754-8411
pISSN - 1754-8403
DOI - 10.1242/dmm.007278
Subject(s) - pten , kras , endometriosis , cancer research , biology , cancer , ovarian cancer , epigenetics , malignant transformation , medicine , pathology , pi3k/akt/mtor pathway , gene , apoptosis , genetics , colorectal cancer
Endometriosis, a major contributor to pelvic pain and subfertility, affects 6–10% of women of reproductive age and is characterized by endometrial-like tissue outside the uterus, mainly in the pelvic peritoneum and ovaries ([Giudice, 2010][1]). It is becoming widely accepted that endometriosis,
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