Open AccessHeterozygous Mutation within a Kinase-Conserved Motif of the Insulin-Like Growth Factor I Receptor Causes Intrauterine and Postnatal Growth Retardation
Author(s) -
Tassilo Kruis,
Jürgen Klammt,
Assimina GalliΤsinopoulou,
Tillmann Wallborn,
Marina Schlicke,
Eva Müller,
Jürgen Kratzsch,
Antje Körner,
Rasha Odeh,
Wieland Kieß,
Roland Pfäffle
Publication year - 2010
Publication title -
molecular endocrinology
Language(s) - English
Resource type - Journals
eISSN - 1944-9917
pISSN - 0888-8809
DOI - 10.1210/mend.24.2.9996
Subject(s) - biology , insulin like growth factor 1 receptor , autophosphorylation , endocrinology , insulin like growth factor , insulin receptor , medicine , protein kinase b , mutation , mapk/erk pathway , microbiology and biotechnology , genetics , kinase , protein kinase a , receptor , signal transduction , growth factor , gene , insulin , insulin resistance
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